基于高尿酸诱导的NRK-52E细胞模型探讨ZAG与 ERK1/2和p38信号通路的交互作用
陈柄源，杨 洁，樊凯璇，陈秉朴
Investigation of the Interaction of ZAG with ERK1/2 and p38 Signaling Pathways Based on the NRK-52E Cell Model Induced by High Uric Acid
CHEN Bingyuan, YANG Jie, FAN Kaixuan, CHEN Bingpu
激光生物学报
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2024, (2): 176
-184
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